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1 General aspects (0)   1.1 Epidemiology (9)   1.2 Population genetics (3)   1.3 Pathogenesis (0)   1.4 Quality of life (3)   1.5 Glaucomas as cause of blindness (6)   1.6 Prevention and screening (4) 2 Anatomical structures in glaucoma (0)   2.1 Conjunctiva (0)   2.2 Cornea (19)   2.3 Sclera (2)   2.4 Anterior chamber angle (2)   2.5 Meshwork (0)     2.5.1 Trabecular meshwork (7)     2.5.2 Schlemms canal (2)     2.5.3 Scleral outlet channels (0)   2.6 Aqueous humor dynamics (0)     2.6.1 Production (0)     2.6.2 Outflow (0)       2.6.2.1 Trabecular meshwork (0)       2.6.2.2 Uveoscleral (0)     2.6.3 Compostion (3)   2.7 Episcleral veins and venous pressure (0)   2.8 Iris (1)   2.9 Ciliary body (4)   2.10 Lens (1)   2.11 Vitreous body (0)   2.12 Choroid, peripapillary choroid, peripapillary atrophy (4)   2.13 Retina and retinal nerve fibre layer (24)   2.14 Optic disc (17)   2.15 Optic nerve (4)   2.16 Chiasma and retrochiasmal central nervous system (1)   2.17 Stem cells (2)   2.20 Other (0) 3 Laboratory methods (0)   3.1 Microscopy (0)   3.2 Electron microscopy (0)   3.3 Immunohistochemistry (5)   3.4 Molecular genetics (0)     3.4.1 Linkage studies (5)     3.4.2 Gene studies (19)   3.5 Molecular biology incl. 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associated with intraocular tumors (0)     9.4.9 Glaucomas associated with elevated episcleral venous pressure (1)       9.4.10 Glaucomas associated with hemorrhage (1)     9.4.11 Glaucomas following intraocular surgery (1)       9.4.11.1 Ciliary block (malignant) glaucoma (0)       9.4.11.2 Glaucomas in aphakia and pseudophakia (3)       9.4.11.3 Epithelial, fibrous, and endothelial proliferation (1)       9.4.11.4 Glaucomas associated with corneal surgery (0)       9.4.11.5 Glaucomas associated with vitreoretinal surgery (2)     9.4.15 Glaucoma in relation to systemic disease (9)     9.4.20 Other (2) 10 Differential diagnosis e.g. anterior and posterior ischemic optic neuropathy (3) 11 Medical treatment (0)   11.1 General management, indication (6)   11.2 Cholinergic drugs (0)   11.3 Adrenergic drugs (0)     11.3.1 Epinephrine (0)     11.3.2 Thymoxamine, dapiprazole (0)     11.3.3 Apraclonidine, brimonidine (1)     11.3.4 Betablocker (4)     11.3.5 Other (0)   11.4 Prostaglandins 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Miscellaneous (5)

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Abstract #18207 Published in IGR 9-2

Blood groups and open-angle glaucoma in Tunisia

Jeddi Blouza A; Loukil I; Mhenni A; Ben Rayana C; Hmida S
Journal Français d'Ophtalmologie 2007; 30: 493-496

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PURPOSE: To determine whether blood groups are genetic markers for primary open angle glaucoma (POAG). METHODS: ABO, rhesus, and Kell and Duffy blood groups were analyzed in 114 POAG cases and in a control group of age- and sex-matched patients (96 cases). RESULTS: AB groups are significantly more frequent in POAG cases (10.5%) than in the control group (2%). However, no association was found between POAG and ABO, rhesus, and Kell and Duffy blood groups even when men and women were studied separately. CONCLUSION: AB blood groups seem to be genetic markers of POAG but further studies are needed to confirm this association. LA: French

Dr. A. Jeddi Blouza, Service d'Ophtalmologie, Hôpital La Rabta, Tunis, Tunisia

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Classification:

1.1 Epidemiology (Part of: 1 General aspects)

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