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1 General aspects (0)   1.1 Epidemiology (16)   1.2 Population genetics (1)   1.3 Pathogenesis (1)   1.4 Quality of life (10)   1.5 Glaucomas as cause of blindness (4)   1.6 Prevention and screening (7) 2 Anatomical structures in glaucoma (0)   2.1 Conjunctiva (7)   2.2 Cornea (18)   2.3 Sclera (7)   2.4 Anterior chamber angle (1)   2.5 Meshwork (0)     2.5.1 Trabecular meshwork (4)     2.5.2 Schlemms canal (1)     2.5.3 Scleral outlet channels (1)   2.6 Aqueous humor dynamics (0)     2.6.1 Production (2)     2.6.2 Outflow (2)       2.6.2.1 Trabecular meshwork (7)       2.6.2.2 Uveoscleral (0)     2.6.3 Compostion (2)   2.7 Episcleral veins and venous pressure (1)   2.8 Iris (5)   2.9 Ciliary body (1)   2.10 Lens (1)   2.11 Vitreous body (0)   2.12 Choroid, peripapillary choroid, peripapillary atrophy (1)   2.13 Retina and retinal nerve fibre layer (20)   2.14 Optic disc (13)   2.15 Optic nerve (3)   2.16 Chiasma and retrochiasmal central nervous system (5)   2.17 Stem cells (1)   2.20 Other (0) 3 Laboratory methods (0)   3.1 Microscopy (2)   3.2 Electron microscopy (2)   3.3 Immunohistochemistry (3)   3.4 Molecular genetics (0)     3.4.1 Linkage studies (0)     3.4.2 Gene studies (13)   3.5 Molecular biology incl. 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associated with intraocular tumors (2)     9.4.9 Glaucomas associated with elevated episcleral venous pressure (0)       9.4.10 Glaucomas associated with hemorrhage (0)     9.4.11 Glaucomas following intraocular surgery (0)       9.4.11.1 Ciliary block (malignant) glaucoma (0)       9.4.11.2 Glaucomas in aphakia and pseudophakia (3)       9.4.11.3 Epithelial, fibrous, and endothelial proliferation (0)       9.4.11.4 Glaucomas associated with corneal surgery (2)       9.4.11.5 Glaucomas associated with vitreoretinal surgery (2)     9.4.15 Glaucoma in relation to systemic disease (18)     9.4.20 Other (0) 10 Differential diagnosis e.g. anterior and posterior ischemic optic neuropathy (7) 11 Medical treatment (0)   11.1 General management, indication (3)   11.2 Cholinergic drugs (0)   11.3 Adrenergic drugs (0)     11.3.1 Epinephrine (0)     11.3.2 Thymoxamine, dapiprazole (0)     11.3.3 Apraclonidine, brimonidine (2)     11.3.4 Betablocker (8)     11.3.5 Other (0)   11.4 Prostaglandins 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    12.8.1 Without tube implant (9)     12.8.2 With tube implant or other drainage devices (14)     12.8.3 Non-perforating (10)     12.8.4 Using laser (0)     12.8.5 Other (0)     12.8.10 Woundhealing antifibrosis (12)     12.8.11 Complications, endophthalmitis (6)   12.9 Trabeculotomy, goniotomy (3)   12.10 Cyclodestruction (2)   12.11 Cyclodialysis (0)   12.12 Cataract extraction (0)     12.12.1 Intracapsular (0)     12.12.2 Extracapsular (0)     12.12.3 Phacoemulsification (2)   12.14 Combined cataract extraction and glaucoma surgery (0)     12.14.1 Intracapsular (0)     12.14.2 Extracapsular (2)     12.14.3 Phacoemulsification (14)   12.15 Capsulotomy (0)   12.16 Vitrectomy (0)   12.17 Anesthesia (2)   12.20 Other (3) 13 Therapeutic prognosis and outcome (0)   13.1 Prognostic factors (0)   13.2 Outcome (0)     13.2.1 IOP (1)     13.2.2 Visual field (0)       13.2.2.1 Progression (1)       13.2.2.2 Improvement (0)     13.2.3 Other (0) 14 Costing studies; pharmacoeconomics (5) 15 Miscellaneous (25)

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Abstract #23009 Published in IGR 11-1

Research on the prevention effect of low molecular weight heparin on inflammatory response after glaucoma with cataract combined with introcular lens implantation

Du J-Y; Wang X-L; Zhao J-F; Guan X-R; Chen X-Q
International Journal of Ophthalmology 2008; 8: 2332-2333

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AIM: To research the prevention effect of low molecular weight heparin (LMWH) on inflammatory response after glaucoma with cataract combined with introcular lens implantation (triplex operations). METHODS: Fifty-two glaucoma cases combined with cataract were randomly divided into 2 groups and were treated with medicine blindly. During triplex operations, control group was given 9g/L NaCl injection and LMWH group received 9g/L NaCl injection adding low molecular weight heparin calcium injection 5U/mL. Postoperative day 2, 4 and 7, the condition of intraocular fibrin exudation, membrane formation and acqueous opacity were observed. RESULTS: On day 2, 4 and 7 after surgery, the degree of intraocular fibrin exudation, membrane formation and acqueous opacity in LMWH group was lighter then that in control group (P < 0.05). CONCLUSION: LMWH can effectively inhibit inflammatory response in eyes after triplex operations. LA: Chinese

Dr. J.-Y. Du, Department of Ophthalmology, First People's Hospital of Xianyang City, Xianyang 712000 Shaanxi Province, China. jy02524200@163.com

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Classification:

12.14.3 Phacoemulsification (Part of: 12 Surgical treatment > 12.14 Combined cataract extraction and glaucoma surgery)

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