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1 General aspects (0)   1.1 Epidemiology (16)   1.2 Population genetics (1)   1.3 Pathogenesis (1)   1.4 Quality of life (7)   1.5 Glaucomas as cause of blindness (7)   1.6 Prevention and screening (9) 2 Anatomical structures in glaucoma (0)   2.1 Conjunctiva (5)   2.2 Cornea (21)   2.3 Sclera (3)   2.4 Anterior chamber angle (7)   2.5 Meshwork (0)     2.5.1 Trabecular meshwork (5)     2.5.2 Schlemms canal (2)     2.5.3 Scleral outlet channels (0)   2.6 Aqueous humor dynamics (3)     2.6.1 Production (0)     2.6.2 Outflow (0)       2.6.2.1 Trabecular meshwork (0)       2.6.2.2 Uveoscleral (1)     2.6.3 Compostion (2)   2.7 Episcleral veins and venous pressure (0)   2.8 Iris (7)   2.9 Ciliary body (1)   2.10 Lens (0)   2.11 Vitreous body (2)   2.12 Choroid, peripapillary choroid, peripapillary atrophy (2)   2.13 Retina and retinal nerve fibre layer (25)   2.14 Optic disc (20)   2.15 Optic nerve (2)   2.16 Chiasma and retrochiasmal central nervous system (2)   2.17 Stem cells (1)   2.20 Other (1) 3 Laboratory methods (0)   3.1 Microscopy (0)   3.2 Electron microscopy (0)   3.3 Immunohistochemistry (1)   3.4 Molecular genetics (1)     3.4.1 Linkage studies (2)     3.4.2 Gene studies (25)   3.5 Molecular biology incl. 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Miscellaneous (18)

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Abstract #26518 Published in IGR 12-3

Clinical features of hypoperfusion retinopathy complicating neovascular glaucoma

Zhou B; Wei S
Neuro-Ophthalmology 2010; 34: 279

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AIM: To analyze the Clinical features of hypoperfusion retinopathy complicating neovascular glaucoma. METHODS: 15 cases (15 eyes) with hypoferfusion retinopathy were retrospectively analyzed. All patients were examined by visual acuity and fundus examination fundus fluorescein angiography (FFA) colour Doppler flow imaging (CDFI) magnetic resonance angiography (MRA) digital subtraction angiography DSA etc. RESULTS: There were 12 cases male and 3 cases female; right eye 10,left eye 5. The age of these patients ranged from 18-78 years old and averaged 40years old. The visual acuity HM 0.8. Posterior segment manifestations included midperipheral intraretinal hemorrhages, narrowed retinal arteries, and neovascularization of the disc and/or retina. All eyes have either ipsilateral common carotid artery obstruction or severe bilateral obstruction of the internal carotid arteries. CONCLUSION: Neovascular glaucoma is a rare and severe complication of hypoperfusion retinopathy. The appearance in fundus and FFA of hypoperfusion retinopathy complicating neovascular glaucoma shows a characteristic clinical feature.

B. Zhou. China PLA General Hospital, BeijingChina.

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Classification:

9.4.5.1 Neovascular glaucoma (Part of: 9 Clinical forms of glaucomas > 9.4 Glaucomas associated with other ocular and systemic disorders > 9.4.5 Glaucomas associated with disorders of the retina, choroid and vitreous)
6.11 Bloodflow measurements (Part of: 6 Clinical examination methods)
6.10.2 Posterior segment (Part of: 6 Clinical examination methods > 6.10 Fluorescein (ICG) angiography)

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