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A 78-year-old male with familial amyloidotic polyneuropathy type I (Met30), presented with rubeotic glaucoma nine months following an uncomplicated vitrectomy for vitreous amyloidosis. There was retinal neovascularization and extensive retinal vascular closure. In the preceding nine months, episodes of 'uveitis' and high intraocular pressure were thought to be due to amyloid protein released into the aqueous leading to trabecular meshwork obstruction and high intraocular pressures, thus compounding the ocular ischemia created by amyloid vascular closure. The patient underwent panretinal photocoagulation and Molteno implant surgery. The rubeosis regressed and pressure control was gained but sight was lost.
Dr. A.A. Dunlop, The Eye Associates, Sydney, New South Wales, Australia
9.4.5.1 Neovascular glaucoma (Part of: 9 Clinical forms of glaucomas > 9.4 Glaucomas associated with other ocular and systemic disorders > 9.4.5 Glaucomas associated with disorders of the retina, choroid and vitreous)
9.4.15 Glaucoma in relation to systemic disease (Part of: 9 Clinical forms of glaucomas > 9.4 Glaucomas associated with other ocular and systemic disorders)