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1 General aspects (0)   1.1 Epidemiology (19)   1.2 Population genetics (0)   1.3 Pathogenesis (5)   1.4 Quality of life (12)   1.5 Glaucomas as cause of blindness (13)   1.6 Prevention and screening (10) 2 Anatomical structures in glaucoma (0)   2.1 Conjunctiva (4)   2.2 Cornea (20)   2.3 Sclera (10)   2.4 Anterior chamber angle (8)   2.5 Meshwork (0)     2.5.1 Trabecular meshwork (9)     2.5.2 Schlemms canal (1)     2.5.3 Scleral outlet channels (0)   2.6 Aqueous humor dynamics (0)     2.6.1 Production (1)     2.6.2 Outflow (0)       2.6.2.1 Trabecular meshwork (5)       2.6.2.2 Uveoscleral (1)     2.6.3 Compostion (1)   2.7 Episcleral veins and venous pressure (1)   2.8 Iris (2)   2.9 Ciliary body (1)   2.10 Lens (0)   2.11 Vitreous body (0)   2.12 Choroid, peripapillary choroid, peripapillary atrophy (3)   2.13 Retina and retinal nerve fibre layer (25)   2.14 Optic disc (22)   2.15 Optic nerve (2)   2.16 Chiasma and retrochiasmal central nervous system (2)   2.17 Stem cells (3)   2.20 Other (0) 3 Laboratory methods (0)   3.1 Microscopy (3)   3.2 Electron microscopy (1)   3.3 Immunohistochemistry (2)   3.4 Molecular genetics (0)     3.4.1 Linkage studies (1)     3.4.2 Gene studies (28)   3.5 Molecular biology incl. 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    9.4.1 Steroid-induced glaucoma (6)     9.4.2 Glaucomas associated with disorders of the cornea, conjunctiva, sclera (1)       9.4.2.1 Iridocorneal endothelial syndrome (ICE, incl. irisatrophy) (2)       9.4.2.2 Posterior polymorphous dystrophy (0)       9.4.2.3 Fuchs' endothelial dystrophy (0)       9.4.2.5 Other (1)     9.4.3 Glaucomas associated with disorders of the iris and ciliary body (0)       9.4.3.1 Pigmentary glaucoma (0)       9.4.3.5 Other (0)     9.4.4 Glaucomas associated with disorders of the lens (0)       9.4.4.1 Exfoliation syndrome (10)       9.4.4.2 Glaucomas associated with cataracts (2)       9.4.4.3 Glaucomas associated with lens dislocation (0)       9.4.4.5 Other (1)     9.4.5 Glaucomas associated with disorders of the retina, choroid and vitreous (0)       9.4.5.1 Neovascular glaucoma (5)       9.4.5.5 Other (9)     9.4.6 Glaucomas associated with inflammation, uveitis (6)     9.4.7 Glaucomas associated with ocular trauma (5)     9.4.8 Glaucomas associated with intraocular tumors (0)     9.4.9 Glaucomas associated with elevated episcleral venous pressure (0)       9.4.10 Glaucomas associated with hemorrhage (4)     9.4.11 Glaucomas following intraocular surgery (0)       9.4.11.1 Ciliary block (malignant) glaucoma (0)       9.4.11.2 Glaucomas in aphakia and pseudophakia (10)       9.4.11.3 Epithelial, fibrous, and endothelial proliferation (0)       9.4.11.4 Glaucomas associated with corneal surgery (7)       9.4.11.5 Glaucomas associated with vitreoretinal surgery (4)     9.4.15 Glaucoma in relation to systemic disease (16)     9.4.20 Other (0) 10 Differential diagnosis e.g. anterior and posterior ischemic optic neuropathy (1) 11 Medical treatment (0)   11.1 General management, indication (5)   11.2 Cholinergic drugs (0)   11.3 Adrenergic drugs (0)     11.3.1 Epinephrine (0)     11.3.2 Thymoxamine, dapiprazole (0)     11.3.3 Apraclonidine, brimonidine (2)     11.3.4 Betablocker (5)     11.3.5 Other (0)   11.4 Prostaglandins (32)   11.5 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Without tube implant (8)     12.8.2 With tube implant or other drainage devices (16)     12.8.3 Non-perforating (3)     12.8.4 Using laser (0)     12.8.5 Other (1)     12.8.10 Woundhealing antifibrosis (16)     12.8.11 Complications, endophthalmitis (9)   12.9 Trabeculotomy, goniotomy (4)   12.10 Cyclodestruction (1)   12.11 Cyclodialysis (1)   12.12 Cataract extraction (0)     12.12.1 Intracapsular (0)     12.12.2 Extracapsular (0)     12.12.3 Phacoemulsification (4)   12.14 Combined cataract extraction and glaucoma surgery (0)     12.14.1 Intracapsular (0)     12.14.2 Extracapsular (0)     12.14.3 Phacoemulsification (3)   12.15 Capsulotomy (0)   12.16 Vitrectomy (0)   12.17 Anesthesia (4)   12.20 Other (1) 13 Therapeutic prognosis and outcome (0)   13.1 Prognostic factors (0)   13.2 Outcome (0)     13.2.1 IOP (1)     13.2.2 Visual field (0)       13.2.2.1 Progression (0)       13.2.2.2 Improvement (0)     13.2.3 Other (0) 14 Costing studies; pharmacoeconomics (14) 15 Miscellaneous (15)

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Abstract #47717 Published in IGR 13-4

Prevalence of cataract surgery and postoperative visual outcome in rural central India: Central India Eye and Medical Study

Nangia V; Jonas JB; Gupta R; Khare A; Sinha A
Journal of Cataract and Refractive Surgery 2011; 37: 1932-1938

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Purpose: To determine the prevalence of cataract surgery and the postoperative visual outcomes in rural central India. Setting: Villages in rural central India. Design: Cohort study. Methods: The population-based Central India Eye and Medical Study examined the prevalence of surgical pseudophakia/aphakia, the postoperative visual acuity, and the reasons for decreased postoperative vision in subjects aged 30+ in rural central India. Visual impairment was defined as (1) presenting visual acuity worse than 6/18 or (2) corrected distance visual acuity (CDVA) worse than 6/18. Results: Of the 9392 eyes (99.7%) of 4711 subjects with available data on the lens status, 318 eyes (3.4%) (234 patients, 129 women) had had cataract surgery (5.0% (plus or minus) 0.3%). Cataract surgery was significantly associated with age (P<.001), female sex (P=.008), shorter axial length (P<.001), and diabetes mellitus (P<.001). The prevalence of postoperative presenting visual impairment was 63% (201/318 eyes) and of postoperative CDVA impairment, 36% (117/318 eyes). The major cause of the former was incorrect intraocular lens (IOL) power (42%); the major causes of the latter were posterior capsule opacification (24%), surgical complications (21%), age-related macular degeneration (10.3%), other macular disorders (4.3%), corneal opacities (3.4%), and glaucoma (2.0%). Surgical complications were significantly more common in the aphakic group than in the pseudophakic group (46.4% versus 2.0%; P<.001). Conclusions: Approximately 5% of the central India population aged 30 years or older had had cataract surgery. Postoperative visual impairment was present in 2 of 3 eyes. The major reasons were incorrect IOL power and surgical complications. Improved IOL power calculations and improved surgical techniques may markedly improve postoperative outcome. Financial Disclosure: No author has a financial or proprietary interest in any material or method mentioned. (copyright) 2011 ASCRS and ESCRS.

V. Nangia. Suraj Eye Institute, Mecosabag Fly Over, Nagpur, 440001, India. Email: nagpursuraj@gmail.com

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Classification:

9.4.11.2 Glaucomas in aphakia and pseudophakia (Part of: 9 Clinical forms of glaucomas > 9.4 Glaucomas associated with other ocular and systemic disorders > 9.4.11 Glaucomas following intraocular surgery)
1.1 Epidemiology (Part of: 1 General aspects)

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