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Abstract #8267 Published in IGR 4-3
Ciliochoroidal effusion induced by topical latanoprost in a patient with Sturge-Weber syndrome
Sakai H; Sakima N; Nakamura Y; Hayakawa K; Sawaguchi SJapanese Journal of Ophthalmology 2002; 46: 553-555
BACKGROUND: To report drug-induced ciliochoroidal effusion in a patient with Sturge-Weber syndrome. CASE: A 17-year-old male presented with unilateral glaucoma associated with Sturge-Weber syndrome. OBSERVATIONS: His corrected visual acuity was RE 20/20 and LE 40/60. Intraocular pressure (IOP) readings by goldmann applanation tonometry were: RE 32 mmHg and LE 12 mmHg. Fundus examination showed marked glaucomatous disc cupping in his right eye and normal findings in his left. The patient had a port-wine stain on his right upper eyelid ipsilateral to the glaucomatous eye. Antiglaucomatous medications were started, including topical latanoprost, with a diagnosis of juvenile onset glaucoma associated with Sturge-Weber syndrome. Ultrasound biomicroscopy showed a 360° circumference ciliochoroidal effusion. Forty days after starting medication, latanoprost treated was discontinued. Ten days later, ultrasound biomicroscopy showed total disappearance of the ciliochoroidal effusion. CONCLUSIONS: Interaction of the enhanced uveoscleral outflow with latanoprost in conjunction with elevated episcleral venous pressure may have caused the congestion of the aqueous humor in the supraciliary choroidal space, resulting in the ciliochoroidal effusion.
H. Sakai, MD, Department of Ophthalmology, University of the Ryukyus, Faculty of medicine, 207 Uchara, Nishihara-cho, Okinawa 903-0215, Japan
Classification:
11.4 Prostaglandins (Part of: 11 Medical treatment)
