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POAG is a chronic, progressive optic neuropathy characterised by morphological changes at the optic nerve head and retinal nerve fibre layer in the absence of other ocular disease or congenital anomalies (with / without a raised IOP). With an expected increase in the population and longevity, POAG is likely to become a major cause of ocular morbidity in the developing world. POAG patients have few symptoms in the early stages. Rarely a high IOP may cause a browache. Transient corneal edema from a raised IOP may cause coloured haloes. Patients with advanced damage often have altered vision. However a careful determination of history and physical findings often helps in timely diagnosis. The goal of treatment is to preserve visual function. The decision to treat is individualized depending on the whether the level of IOP will lead to progressive nerve damage. Available treatment algorithms rely on medical management to achieve the target IOP, failing which filtering surgery can be resorted to. In the Indian context, early filtering surgery to acheive the desired target pressure is a viable alternative. Laser trabeculoplasty is an intermediate step. The role of neuroprotection is not yet established clinically.
D. Sood. Glaucoma Imaging Centre, P-13 South Extension Part-II, New Delhi-110049, India.
11.1 General management, indication (Part of: 11 Medical treatment)
12.1 General management, indication (Part of: 12 Surgical treatment)