advertisement

Topcon

Kumar S 10

Showing records 1 to 10 | Display all abstracts from Kumar S

69322 Amacrine cells coupled to ganglion cells via gap junctions are highly vulnerable in glaucomatous mouse retinas
Akopian A
Journal of Comparative Neurology 2016; 0:
69132 Revisiting the role of erythropoietin for treatment of ocular disorders
Shirley Ding SL
Eye 2016; 30: 1293-1309
69322 Amacrine cells coupled to ganglion cells via gap junctions are highly vulnerable in glaucomatous mouse retinas
Kumar S
Journal of Comparative Neurology 2016; 0:
69132 Revisiting the role of erythropoietin for treatment of ocular disorders
Leow SN
Eye 2016; 30: 1293-1309
69322 Amacrine cells coupled to ganglion cells via gap junctions are highly vulnerable in glaucomatous mouse retinas
Ramakrishnan H
Journal of Comparative Neurology 2016; 0:
69132 Revisiting the role of erythropoietin for treatment of ocular disorders
Munisvaradass R
Eye 2016; 30: 1293-1309
69322 Amacrine cells coupled to ganglion cells via gap junctions are highly vulnerable in glaucomatous mouse retinas
Viswanathan S
Journal of Comparative Neurology 2016; 0:
69132 Revisiting the role of erythropoietin for treatment of ocular disorders
Koh EH
Eye 2016; 30: 1293-1309
69322 Amacrine cells coupled to ganglion cells via gap junctions are highly vulnerable in glaucomatous mouse retinas
Bloomfield SA
Journal of Comparative Neurology 2016; 0:
69132 Revisiting the role of erythropoietin for treatment of ocular disorders
Bastion ML; Then KY; Kumar S; Mok PL
Eye 2016; 30: 1293-1309

Issue 18-1

Change Issue


advertisement

Nidek