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Author #40830
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Hu C
27
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Hu C
106364
A Novel and Reversible Experimental Primate Ocular Hypertension Model: Blocking Schlemm's Canal
Jiang J
Ophthalmic Research
2022; 0:
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Shang X
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Lin S
British Journal of Ophthalmology
2024; 108: 188-194
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Wang X
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Shang X
British Journal of Ophthalmology
2024; 108: 188-194
106364
A Novel and Reversible Experimental Primate Ocular Hypertension Model: Blocking Schlemm's Canal
Xu J
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Wang X
British Journal of Ophthalmology
2024; 108: 188-194
106364
A Novel and Reversible Experimental Primate Ocular Hypertension Model: Blocking Schlemm's Canal
Tao Y
Ophthalmic Research
2022; 0:
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Zhou K
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Chu X
British Journal of Ophthalmology
2024; 108: 188-194
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Pan X
Ophthalmic Research
2022; 0:
106364
A Novel and Reversible Experimental Primate Ocular Hypertension Model: Blocking Schlemm's Canal
Hu C
Ophthalmic Research
2022; 0:
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Huang Q
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Hu C
British Journal of Ophthalmology
2024; 108: 188-194
106364
A Novel and Reversible Experimental Primate Ocular Hypertension Model: Blocking Schlemm's Canal
Zhang C
Ophthalmic Research
2022; 0:
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Chu X
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Si Y
British Journal of Ophthalmology
2024; 108: 188-194
106364
A Novel and Reversible Experimental Primate Ocular Hypertension Model: Blocking Schlemm's Canal
Sun X; Ye C
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Chen DF
British Journal of Ophthalmology
2024; 108: 188-194
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Hu C
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Zhou W
British Journal of Ophthalmology
2024; 108: 188-194
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Xu X
Ophthalmic Research
2022; 0:
106364
A Novel and Reversible Experimental Primate Ocular Hypertension Model: Blocking Schlemm's Canal
Zhang S
Ophthalmic Research
2022; 0:
106362
Faster Macular Vessel Density Loss in More Advanced Primary Open Angle Glaucoma Eyes
Liang Y
Ophthalmic Research
2022; 0:
106747
Decreased macular deep capillary plexus is associated with functional progression of normal tension glaucoma patients with unilateral visual field loss
Kong YXG
British Journal of Ophthalmology
2024; 108: 188-194
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