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Sato K 22
Showing records 1 to 22 | Display all abstracts from Sato K109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Sato K
GLIA 2023; 71: 2609-2622
109753 DPP-4 Inhibitors Attenuate Fibrosis After Glaucoma Filtering Surgery by Suppressing the TGF-β/Smad Signaling Pathway
Yoshida M
Investigative Ophthalmology and Visual Science 2023; 64: 2
109639 Reduced Plasma BDNF Levels in Normal Tension Glaucoma Compared to Open Angle Glaucoma
Sato K; Takada N
Journal of Glaucoma 2023; 32: 734-737
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Ohno-Oishi M
GLIA 2023; 71: 2609-2622
109753 DPP-4 Inhibitors Attenuate Fibrosis After Glaucoma Filtering Surgery by Suppressing the TGF-β/Smad Signaling Pathway
Kokubun T; Sato K
Investigative Ophthalmology and Visual Science 2023; 64: 2
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Yoshida M
GLIA 2023; 71: 2609-2622
109639 Reduced Plasma BDNF Levels in Normal Tension Glaucoma Compared to Open Angle Glaucoma
Fujioka A; Himori N
Journal of Glaucoma 2023; 32: 734-737
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Sato T
GLIA 2023; 71: 2609-2622
109753 DPP-4 Inhibitors Attenuate Fibrosis After Glaucoma Filtering Surgery by Suppressing the TGF-β/Smad Signaling Pathway
Tsuda S
Investigative Ophthalmology and Visual Science 2023; 64: 2
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Aizawa T
GLIA 2023; 71: 2609-2622
109639 Reduced Plasma BDNF Levels in Normal Tension Glaucoma Compared to Open Angle Glaucoma
Yokoyama Y
Journal of Glaucoma 2023; 32: 734-737
109753 DPP-4 Inhibitors Attenuate Fibrosis After Glaucoma Filtering Surgery by Suppressing the TGF-β/Smad Signaling Pathway
Yokoyama Y; Himori N
Investigative Ophthalmology and Visual Science 2023; 64: 2
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Sasaki Y
GLIA 2023; 71: 2609-2622
109639 Reduced Plasma BDNF Levels in Normal Tension Glaucoma Compared to Open Angle Glaucoma
Tsuda S
Journal of Glaucoma 2023; 32: 734-737
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Maekawa S
GLIA 2023; 71: 2609-2622
109753 DPP-4 Inhibitors Attenuate Fibrosis After Glaucoma Filtering Surgery by Suppressing the TGF-β/Smad Signaling Pathway
Nakazawa T
Investigative Ophthalmology and Visual Science 2023; 64: 2
109639 Reduced Plasma BDNF Levels in Normal Tension Glaucoma Compared to Open Angle Glaucoma
Omodaka K; Kirihara T
Journal of Glaucoma 2023; 32: 734-737
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Ishikawa M; Omodaka K
GLIA 2023; 71: 2609-2622
109639 Reduced Plasma BDNF Levels in Normal Tension Glaucoma Compared to Open Angle Glaucoma
Ishikawa M; Kunikata H
Journal of Glaucoma 2023; 32: 734-737
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Kawano C
GLIA 2023; 71: 2609-2622
109639 Reduced Plasma BDNF Levels in Normal Tension Glaucoma Compared to Open Angle Glaucoma
Nakazawa T
Journal of Glaucoma 2023; 32: 734-737
109891 The GPR84 molecule is a mediator of a subpopulation of retinal microglia that promote TNF/IL-1α expression via the rho-ROCK pathway after optic nerve injury
Ohue-Kitano R; Kimura I; Nakazawa T
GLIA 2023; 71: 2609-2622
