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22-1
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Classification #3.6
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3.6 Cellular biology
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3.6 Cellular biology
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92166
Influence of Anti-Glaucoma Drugs on Uptake of Extracellular Vesicles by Trabecular Meshwork Cells
Tabak S
International Journal of Nanomedicine
2021; 16: 1067-1081
92473
Molecular Insights into Myocilin and Its Glaucoma-Causing Misfolded Olfactomedin Domain Variants
Lieberman RL
Accounts of chemical research
2021; 54: 2205-2215
92065
Glial Cells in Glaucoma: Friends, Foes, and Potential Therapeutic Targets
García-Bermúdez MY
Frontiers in neurology
2021; 12: 624983
92021
Oxidative Stress and the Role of NADPH Oxidase in Glaucoma
Fan Gaskin JC
Antioxidants (Basel, Switzerland)
2021; 10:
92516
Extracellular Matrix Remodeling in the Retina and Optic Nerve of a Novel Glaucoma Mouse Model
Reinhard J
Biology
2021; 10:
92123
Mitochondrial function is impaired in the primary visual cortex in an experimental glaucoma model
Hvozda Arana AG
Archives of Biochemistry and Biophysics
2021; 701: 108815
92025
Role of Choline in Ocular Diseases
Hwang JS
International journal of molecular sciences
2021; 22:
92074
Crosstalk between MicroRNA and Oxidative Stress in Primary Open-Angle Glaucoma
Tabak S
International journal of molecular sciences
2021; 22:
92031
Cannabidiol and the Canonical WNT/β-Catenin Pathway in Glaucoma
Vallée A
International journal of molecular sciences
2021; 22:
92745
Adenosine A3 receptor activated in HO oxidative stress of primary open-angle glaucoma
Zhou Z
Annals of translational medicine
2021; 9: 526
92126
Identification of key genes and pathways in scleral extracellular matrix remodeling in glaucoma: Potential therapeutic agents discovered using bioinformatics analysis
Hu D
International journal of medical sciences
2021; 18: 1554-1565
92822
Trabecular meshwork's collagen network formation is inhibited by non-pigmented ciliary epithelium-derived extracellular vesicles
Tabak S
Journal of cellular and molecular medicine
2021; 25: 3339-3347
92545
A novel ocular function for decorin in the aqueous humor outflow
Schneider M
Matrix Biology
2021; 97: 1-19
92166
Influence of Anti-Glaucoma Drugs on Uptake of Extracellular Vesicles by Trabecular Meshwork Cells
Tabak S
International Journal of Nanomedicine
2021; 16: 1067-1081
92728
A Porcine Organ-Culture Glaucoma Model Mimicking Trabecular Meshwork Damage Using Oxidative Stress
Snider EJ
Investigative Ophthalmology and Visual Science
2021; 62: 18
92173
Role of Retinal Amyloid-β in Neurodegenerative Diseases: Overlapping Mechanisms and Emerging Clinical Applications
Wang L
International journal of molecular sciences
2021; 22:
92866
Activation of ATF4 triggers trabecular meshwork cell dysfunction and apoptosis in POAG
Ying Y
Aging
2021; 13: 8628-8642
92290
Silencing microRNA‑29b‑3p expression protects human trabecular meshwork cells against oxidative injury via upregulation of RNF138 to activate the ERK pathway
Liu H
International Journal of Molecular Medicine
2021; 47:
92074
Crosstalk between MicroRNA and Oxidative Stress in Primary Open-Angle Glaucoma
Tabak S
International journal of molecular sciences
2021; 22:
92138
Retinal Molecular Changes Are Associated with Neuroinflammation and Loss of RGCs in an Experimental Model of Glaucoma
Fernández-Albarral JA
International journal of molecular sciences
2021; 22:
92822
Trabecular meshwork's collagen network formation is inhibited by non-pigmented ciliary epithelium-derived extracellular vesicles
Tabak S
Journal of cellular and molecular medicine
2021; 25: 3339-3347
92360
Lithium and Atypical Antipsychotics: The Possible WNT/β Pathway Target in Glaucoma
Vallée A
Biomedicines
2021; 9:
92575
Oxidative Stress and Hypoxia Modify Mitochondrial Homeostasis During Glaucoma
Jassim AH
Antioxidants & redox signaling
2021; 35: 1341-1357
92458
MiR-137 promotes cell growth and inhibits extracellular matrix protein expression in HO-induced human trabecular meshwork cells by targeting Src
Wang L
Neuroscience Letters
2021; 755: 135902
92132
New solutions for old challenges in glaucoma treatment: is taurine an option to consider?
Iezhitsa I
Neural Regeneration Research
2021; 16: 967-971
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